○ Experience
• (2017-present) 성균관대학교 의과대학 교수
• (2011-2017) 성균관대학교 의과대학 부교수
• (2010-2011) 미국 The Johns Hopkins University, Visiting Professor
• (2007-2011) 성균관대학교 의과대학 조교수
• (2004-2007) 강원대학교 의과대학 전임강사
• (2003-2004) 서울대학교 의과대학 Postdoctoral fellow
• (2003) 서울대학교 의과대학 의학박사 (생리학)
• (1997) 서울대학교 의과대학 의학사
○ Major Research Achievements
1) Yoon JY, Ho WK, Kim ST, Cho H. Constitutive CaMKII activity regulates Na+ channel in rat ventricular myocytes. J Mol Cell Cardiol. 2009, in Press.
2) Cho H, Kim YA, Ho WK. Phosphate number and acyl chain length determine the subcellular location and lateral mobility of phosphoinositides. Mol Cells. 2006, 22(1): 97-103.
3) Cho H, Kim YA, Yoon JY, Lee D, Kim JH, Lee SH, Ho WK. Low mobility of phosphatidylinositol 4,5-bisphosphate underlies receptor specificity of Gq-mediated ion channel regulation in atrial myocytes. Proc Natl Acad Sci U S A. 2005, 102(42):15241-6.
4) Cho H, Lee SH, Lee D, Ho WK. Receptor-induced depletion of phosphatidylinositol 4,5-bisphosphate inhibits inwardly rectifying K+ channels in a receptor-specific manner. Proc Natl Acad Sci U S A. 2005, 102(12):4643-8.
5) Cho H, Hwang JY, Kim D, Shin HS, Kim Y, Earm YE, Ho WK. Acetylcholine-induced phosphatidylinositol 4,5-bisphosphate depletion does not cause short-term desensitization of G protein-gated inwardly rectifying K+ current in mouse atrial myocytes. J Biol Chem. 2002, 277(31):27742-7.
6) Cho H, Nam GB, Lee SH, Earm YE, Ho WK.Phosphatidylinositol 4,5-bisphosphate is acting as a signal molecule in alpha(1)-adrenergic pathway via the modulation of acetylcholine-activated K(+) channels in mouse atrial myocytes. J Biol Chem. 2001, 276(1):159-64.